Topical Blog Week #6 (Due Wednesday)

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What we would like you to do is to find a topic from this week's chapter that you were interested in and search the internet for material on that topic. You might, for example, find people who are doing research on the topic, you might find web pages that discuss the topic, you might find a video clip that demonstrates something related to the topic, etc. What you find and use is pretty much up to you at this point. Please be sure to use at least 3 quality resources. If you use videos, please limit it to one video.

Once you have completed your search and explorations we would like you to:
1a) State what your topic is.
1b) Discuss how the topic relates to the chapter.
1c) Discuss why you are interested in it.

2) Next, we would like you to take the information you read or viewed related to your topic, integrate/synthesize it, and then write about the topic in a knowledgeable manner. By integrating/synthesizing we mean taking what your read/experienced from the internet search organize the information into the main themes, issues, info, examples, etc. about your topic and then write about the topic in your own words using the information you have about the topic.

Synthesizing information is hard for some people to do - many students write what we refer to as "serial abstracts." They are tempted to talk about the websites rather than the topic proper Serial abstract writers will talk all about website #1, start a new paragraph and talk all about web site #2, start a new paragraph and talk all about web site #3, and then write some kind of conclusion about the three websites they used. Serial means one after the other...again, this is what you DON'T want to do! We want you to talk about the topic, not the websites!

Also, if all three sites are on the same one topic it will be easier.

At first it is a real challenge to get out of the habit of writing "serial abstracts," but we assure you once you get the hang of it writing about the topic is much easier to do using the integration method. And besides this is the way researchers and scientists write their technical reports and findings - many of you will have to be able to do this for other classes and for jobs that you may eventually be hired for so now is a good time to learn this skill.

At this point don't worry so much about a grade, worry about doing your best to have fun with the topic and then integrate it into your own words to share what you found and what you now know.

3) List the terms you used from the text and from your reference websites.

4) At the end of your post, please include working URLs for the three websites. For each URL you have listed indicate why you chose the site and the extent to which it contributed to your post.

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My topic this week is about the two primary theories of colour perception. This topic is closely connected to the chapter, which discusses what happens when the eyes perceive colours (or different physical wavelength. I am interested in this topic because upon reading the chapter and the research, I wonder which theory is more dominant in explaining colour perception.

THEORIES OF COLOUR PERCEPTION

Why do we see colours differently than other species? Why is there a slight different between the colour perceptions of each individual human? It is hard to believe that the colours we see every day is merely an interaction between the physical properties of our eyes and the external physical stimuli, but it is also “the” interaction between the our visual system and the external stimuli! It sounds just as simple and just as complicated as it sounds. Different theories was developed to explain why we see what we see, and I am discussing the two main theories in this blog post: trichromatic theory and the opponent process theory.

Both trichromatic theory and the opponent process theory aim to discuss how we see colour, or how the interaction between the physical properties of light and our perception of colour in respect to our nervous system. Each theory has its flaws and advantages, and there are plenty of research which can provide ample support for both theories. We will have to judge which one we can go with.

The trichromatic theory, also known as the three colour theory, was first proposed by Thomas Young in the early 19th century, and modified by Hermann von Helmholtz half a century later; therefore it can also be known as the Young Helmholtz theory. The idea of this theory is basically colour matching. The fundamental element of this theory is that our visual system sees three colours, and the further mixing of these three primary colours essentially creates the rest of the colours we see. The three primary colours are red, green, and blue.

The physical structure of our eyes suggest that this theory is can be true. There are three types of colour detecting cones in the retina, and each types of cones can detect different wavelengths. One can detect longer wavelengths that show red, one is sensitive to medium wavelengths that appear green, and the last set of cones reacts to short wavelengths such as blue. These cones are L (long wavelengths), M (medium wavelengths), and S (short wavelengths) cones, respectively.

Besides the anatomy of the eyes, the colour matching experiment Young and Helmholtz developed also supported the theory. The researchers show a primary colour to the participants, who were then asked to adjust the intensities of the mixture of two colours together until the mixture appear as close to the primary colour as possible. Although the mixture colour can never become identical to the primary colour, because of the substantially different physical components, but the participants can make them “appear” identical.

However, this theory cannot explain some of the perception phenomenon, such as after images of colours. Try staring at a red dot for a while before looking at a white wall, where you will see a green dot. Same thing can happen to yellow and blue. Try staring at a blue dot before looking at a white wall, what do you see?

Based upon the after images, Ewald Hering proposed the proponent process theory in the late 19th century. The theory has two basic ideas, colour opponency and adapting. Colour opponency suggests that the colour sensitive cones work together to form three pairs of opposing colours: black and white, blue and yellow, and red and green. Colour perception is thought to occur within these three channels by competing with the opposing colours. For example, when staring at a red dot for so long, the red-sensitive cones get worn out, and the blue-sensitive cones wins over when you move your fixation onto a white wall, and that green dot you see is the after image. Adapting suggests that the interaction between the cones’ coordination of light and the central visual system does not produce fixed colour, which changes under different context where the lights are viewed.

However, this theory seems odd when we think of the colour yellow. There is no yellow-sensitive cones. Yellow is essentially explained with the interactions between red and green wavelengths. When the brain detects more blue activities hitting the retina, we see blue; when the brain detects more activities of the combination or red and green wavelengths hitting the retina, we see yellow. How this theory works is basically how we mix different water colours in grade school – remember how you were amazed with how yellow pops up after you mix green and red together?

Some further research has shown supports for this theory. In the mid-20th century, an experiment consists of electrical recordings from the LGN shows that it is where colour perception takes place – the LGN from the brain. Further studies using modern technology also provide proof that bipolar and ganglion cells are involved in colour perception; the bipolar cells convert information received from the cones, the pass all the information to the ganglion cells where a bunch of colours are being handled. This process is an important part of the opponent process theories.

Both theories have empirical scientific support, but both fail to explain colour constancy. These two theories complement each other, and can be combined to explain how our visual system actually works. The Young Helmholtz theory explains the basis of the physiological reaction to different wavelengths that hit our eyes, and the opposing process theory explains the neurological processing of those physiological information. None can be accounted more than the other.

TERMS: neurological, physical, visual system, LGN, ganglion cells, bipolar cells, cones, wavelengths, trichromatic theory, opposing process theory, colour matching, colour constancy, after images, adapting, colour opponency

http://www.ukessays.co.uk/essays/psychology/theories-of-vision.php

This is a well written essay explaining the basic elements of both t
heories, as well as providing the comparison between the two. I was able to understand a longer research paper after reading this essay, which was the most beneficial website regarding this blog post.

http://color.psych.upenn.edu/brainard/papers/ColorTheory1.pdf

This is a research paper on both theories, where mathematic formulas were involves. I scanned through the paper. It was hard to understand but it provided me with empirical evidence that supports both theories, which I thought was very beneficial as it helped me more in my understanding of how the theories were supported.

http://psych.ucalgary.ca/PACE/VA-Lab/colourperceptionweb/theories.htm

This article was not at all that useful since it provided me with minimal information regarding colour perception, but it was helpful in a sense that it makes the basic components of the two theories a lot easier to understand.

1a) State what your topic is.
My topic is about color vision in animals.

1b) Discuss how the topic relates to the chapter.
as much as visual system is very important in many ways, color matter most too, in this section of the chapter, it relates to our chapter because it explains how certain things work, how certain plants are made for different type of animals, while we think that the forest is colorful and it's something for us to help ourselves in, it was actually made for the animals who live there, a plant that we think is colorful is most likely is, but it's made for it's own animal for a specific reason to attract that particular animal to do what its purpose for it is.
short wavelengths are very usual to bees because it helps them recognize a plant that they can mate in, as in other words, this is called ultraviolet light, this section is related to our chapter because it gives us information from a different perception, the perception of how animals receive colors, why certain plants are made a particular way, and how we can improve our survival skills if we ever come across situations in the deep jungle.
1c) Discuss why you are interested in it.
as i stated before, i was wondering whether or not the animals see the same colors as we do, or they have higher intensity of colors, or maybe someone of them do see the same like us, in other words, some animals just like humans, they have however different photoreceptors, humans have people who can't see colors, and the ones who can, animals kind of have the same system as well. as well as it is, there are some animals who in fact have a higher intensity of colors than we do, humans have trichromats, which mean we have three different types of photoreceptors, while dogs, have dichromats, which mean they only have two photoreceptors, but the very thing that i found interesting is that chickens in fact have tetrachromats which mean they have four layers of photoreceptors. in one of the videos i came across from youtube, dogs don't have the best of vision, in fact their vision is compare to us using night goggles webcam vision, it's greenish and kind of dark in some areas, but cats visual system is the closest to us and is more of the morning light for us, but when it comes to birds, they actually have a better color vision than we do, they see color more intensively and their colors are very bright and vivid and so on and so forth. something that i found really interesting as well is how rats see, rats visions would be like a human vision with a lazy eye, each eyes move on their own but one have more fast movements than the other and the one is literally lazy. i thought these things were very interesting as well, another source that i stumbled upon was also very helpful in helping us understand how different their visual system work too, gecko and a frog are actually a good example of animals who can see colors in a very dim lighting, they can tell the color blue from any other colors, and they can also see the difference in the many greys we have just like a person who isn't color blind can tell the difference. another interesting thing that i thought was very interesting in my findings was how a simple gene can give you a mutation to be color blind or not, whether you're a human or an animals, long ago, our ancestors were able to see many and many more pigments, but as the time passed down, we started to lose more and more each time, which would bring us to our time now, in this being of state, we can only see certain colors, and that's because our mutated gene isn't as dominant as it once was , and this same concept goes for animals as well.

Findings :
https://www.youtube.com/watch?v=6hYaT4gvjNc
This gives you an idea on how different animals have different levels of visions and their vision is built for them to use it to their own advantage.

http://www.colormatters.com/color-matters-for-kids/how-animals-see-color
This source is very helpful on the variations of colors between all kinds of animals.

http://phys.org/news/2012-05-genetic-mutations.html
as I was reading through this article, it was also speaking about how dog can be tested for color blindness, how we used to have different types of pigments for colors millions years ago, but through evolution, we became to only have few of those pigments left.

Terms : Color pigment, trichromats, tetrachromats, dichromats, wavelengths, ultraviolet, color blindness, mutated genes, photoreceptors, visual fields, visual system, cones, rods, color droplets


I chose to research the topic of color blindness and color vision in animals. This was briefly discussed in the chapter but a lot about the different amount of cones and wavelengths that animas are able to see is also discussed and related to the chapter. I choose to research this because I thought it would be interesting, and I always wondered if animals see things the way that we do, or if they see differently.

To begin with, animals are typically tested for color vision by the placement of multiple panels with various colors. Two of the panels are typically the same and the third is different. If they are able to identify the panel that has the varied color, they are rewarded. The panels are then switched up to see if they can still identify the correct color. However, a study was done for goldfish where they had to swim to a correct panel in order to receive the treat. Light intensity and color was changed to see if it would affect their ability to identify the correctly colored panel and it was found that it did not.

Color blindness typically occurs on the X chromosome and is recessive. This means that females get it less because if one X chromosome is healthy then the disease is not present. It also means that men cannot pass color blindness to their sons, because they only pass on their Y chromosome. Most animals are at least partially color blind, but often times the colors animals see are for evolutionary purposes.

Next the different visual systems of several animals were discussed. Insects were discussed several times, mainly bees. Like humans bees have a trichromatic visual system (3 cone classes), but see yellow/blue/ultraviolet instead of red/green/blue. Their eyes also have multiple lenses, which results in low resolution. An interesting fact mentioned was that if we had multiple lenses and kept our resolution, as humans, our eyes would be as big as hula-hoops. Insects are also thought to have more complex eyes because their processing occurs in the eye instead of like the brain as in humans. Butterflies can also see ultraviolet markings in flowers (like bees), but can also identify ultraviolet markings on other butterflies that help them identify each other. Humans cannot usually see these markings, unless they are also iridescent.

Cats are dichromate (2 cones) and are very similar to humans with red-green color blindness. They are thought to have daytime vision that is 6 times worse than humans, and due to a larger amount of rods than cones, night vision that is 6 times better than humans.

Birds are tetrachromats (4 cones) and can see green/red/blue/ultraviolet, and sometimes have better resolution than humans. The ultraviolet helps them identify some animals with ultraviolet markings. They also can see underwater while moving unlike humans. Pigeons are thought to be able to see as many as 5 spectral bands of color and are very good at identifying the difference between colors that resemble each other closely.


Rattlesnakes are unique like other snakes they have a special neural receptor that is able to transform infrared light into neural signals. Interestingly enough, humans have the same neural receptor but ours sends signals of pain after eating spicy foods, instead of interpreting infrared heat signatures. Snakes combine their very low-resolution visual images with the infrared image to create their image of vision.

Cuttlefish were also discussed as being completely unique from vertebrates. They have no blind spot and their pupils are shaped like W’s instead of O’s. They are completely colorblind but have one photoreceptor that allows them to see grey. They are however, able to see polarization, which they use to communicate with other fish.

Terms: color blindness in animals, cones, rods, wavelengths, trichromatic, dichromate, tetrachromats, resolution, ultraviolet, light intensity, color, vision, spectral bands of color, neural receptor, resolution, infrared, photoreceptor, polarization, blind spot

http://nautil.us/issue/11/light/how-animals-see-the-world
I used this website because it included slides that allowed you to see the difference between animal and human vision. It covered a lot of animal’s visions and contributed a lot to my post.

http://www.nytimes.com/1985/08/13/science/vision-through-animal-eyes-reveals-surprising-color.html?pagewanted=2
This website was useful because it talked about how animals are tested for color blindness, as well as basic terminology that was discussed in the book, but it helped me better understand the vocab. It discussed specific experiments done on animals, so it also contributed a lot.

http://www.webexhibits.org/causesofcolor/17C.html
This website was reviewing a lot from the other websites I had visited but added information on insects and butterflies that I hadn’t read before as well as other animals. It contributed the least but was still very informative.

1a) State what your topic is.
Color vision in animals and color blindness.

1b) Discuss how the topic relates to the chapter.
This relates to the chapter because the chapter went into depth on both of these topics. But I fel that they relate to each other. The chapter talked about the different cones and wavelengths that animals are able to see. I thought it’d be interesting to learn more on this topic because people try to teach their animals tricks based on color, but I’ve always wondered if the colors they see are different than the ones we describe.

The ability to use wavelength information has evolved several times in several ways during the course of evolution. We’ve been able to adapt orientation vision and motion vision throughout the years. Having this vision makes it easier for us to find food and to have sex, which is essential for animals. Color vision is accomplished in different ways in different species. It’s possible to use a single photopigment to create more than one functional type of cone. We put a different filter in front of each type of cone so that some wavelengths are subtracted before light reaches the photoreceptor. Different species have different lights, and each species’ visual systems appear to be turned to its particular wavelength structure. A combination of photopigment and a colored filter makes signals for conspecifics.

In humans, there are three types of cone cell that are responsible for the early stages of color vision. We are trichromats, with two types of photoreceptors. Our S, M, and L cones are different because they contain different photopigments. Each type of cone cell is maximally absorbent in a different part of the spectrum, which is red, green, and blue cones. When light hits the eye, the cones are stimulated differently according to their type, and the relative excitation of each type underlies color sensation. Animals vary in the number of sensitivity of cones present. So the visual process can result in different color sensations compared to humans. Most animals are dichromatic, which means they only are blue and green sensitive.

Some animals do have considerably good color vision. Monkeys, squirrels, birds, insects, and many fish can see a pretty fairly good range of color. On the other scale is cats and dogs who can’t see that many colors. This is because monkeys and birds and such need to be able to have good color vision to identify between ripe and unripe fruits an foods. Colors also make animals more attractive to each other when they mate. They also need this ability to identify predators.

There’s no real way to know what animals can actually see, but scientists have examined the cones inside the eyes to at least estimate what colors animals can see. Scientists have been able to use microspectrophotometry to analyze the visual pigments and photosensitivity of cells in order to determine what animals can see. They have also used behavioral tests to identify this.

Terms: Color vision in animals, color blindness, cones, wavelengths, orientation vision, motion vision, trichromats, photoreceptors, vision, species, S-cones, M-cones, L-cones, photopigments, colored filter, visual system, filter, light, sensation, dichromatic, microspectrophotometry, photosensitivity, cells.

URL: http://www.iflscience.com/plants-and-animals/inside-colorful-world-animal-vision

URL: http://www.colormatters.com/color-matters-for-kids/how-animals-see-color

URL: https://www.youtube.com/watch?v=6hYaT4gvjNc

I choose these websites because they all had really good info on the topic. The youtube did wonderful at describing visually how they see. The other two websites had really good info that were able to help me expand more and learn more on the topic.

1a. I chose to focus on the visual system and different ways it influences our perception of color regarding the human face, connotations of color, and mate selection in the animal kingdom. I decided to give myself a broad spectrum of research to look at because I wanted to exemplify the idea that perception of color effects every aspect of life and the evolutionary process.
1b. This topic relates to chapter five because it provides context and research studies that demonstrate the importance of our perception of color and the importance of color perception in the animal kingdom. It covers the parts of the visual system associated with perceiving color and studies that provide strong evidence for the effects that color and shading have on our perception and for what our brain finds pleasurable.
1c. I am interested in this topic because it relates to every aspect of what we experience on a daily basis. We are constantly bombarded with physical properties that our brain perceives to have color and shading. The perception facilitated by our brain influences our attraction, our dislike, and our perception of an entire environmental or social situation. Additionally I am interested in how this perception affects the animal kingdom and their mate selection. I think it is worthwhile to discover the extent to which animals perceive objects and color the way we do. I enjoy researching topics like this because it has real world applications and has an affect on so much more than we realize.
2. The first article by Nakajima et al. found that our perception of facial color is vitally important for correctly perceiving the emotion and general health of another person while communicating. When we are correctly able to perceive the emotions of another person a neuron specific for facial recognition fires and allows us to interact in a socially acceptable way. This is relevant because it would be maladaptive to be unable to correctly perceive emotion. In a social context it would cause exclusion if we responded to a persons sad expression with joy and happiness. The ability to be empathetic and to interact healthily with our social groups is heavily influenced by our brains perception of the color and shading of the face. I found this very interesting because I have not often thought about the process my brain goes through when it is interpreting the facial expressions and emotions of someone else. We are typically able to perceive these traits or expressions in a matter of seconds and without much thought. It is incredible to realize that our brains ability to perceive color is directly related to our social interactions with people and our ability to read their emotions accurately. A second article by Sorokowski et al. explored the idea that the color of a sports uniform actually effects sports performance as a result of our brains perception of the color. The researchers found that there is a cross cultural finding that supports cultural relativism and the idea that that the colors red and black are associated with winning. Athletes wearing a different color during a sport that involves competition between two or more players were perceptually found to be weaker and less likely to win, even if that perception was overtly wrong. This finding suggests that color plays an important role in our perception of inferiority in the context of sports. I find this endlessly interesting and have so many questions regarding this finding. It is amazing that colors such as black and red that often indicate negative emotions or situations are perceived as more likely to win when worn by an athlete. One could hypothesize that the use of red and black for traffic lights and clothing worn by important people could result in the colors being associated with power. I found this article to be very relevant to the our perception of color and the effects it has on our daily lives. Lastly, an article by Hughes et al. also demonstrated the importance of certain colors on the perception of monkeys and their mate selections. Generally, humans and animals find possible mates more attractive when they are associated with the color red. To test these findings researchers presented monkeys in their sample with pictures of the rear ends of the opposite sex with different color backgrounds. The results showed that female monkeys were more attracted to the image that was presented with a colorful (red) background. This finding is interesting because it demonstrates the importance of our perception in color. It follows the same concept of the jersey research study because it shows that certain colors are associated with dominance and specifically a fit mate. I think it is incredible that something as small as our perception of the color the opposite sex is wearing can have an effect on our perception of their attractiveness. All of these findings are thought provoking and provide applicable research to understand our perception of color.
Terms: visual system, color perception, perception, sensory, cultural relativism

http://web.b.ebscohost.com.proxy.lib.uni.edu/ehost/detail/detail?vid=6&sid=1301bea3-ba7e-4c02-9e4c-c46b097ed143%40sessionmgr112&hid=109&bdata=JnNpdGU9ZWhvc3QtbGl2ZQ%3d%3d#db=psyh&AN=2014-40934-001 I chose this article because it showed strong evidence for the idea that color perception influences mate choices in animals and also humans.

http://web.b.ebscohost.com.proxy.lib.uni.edu/ehost/detail/detail?vid=7&sid=23aea01e-51d0-4c5e-9941-72e727b5826e%40sessionmgr198&hid=109&bdata=JnNpdGU9ZWhvc3QtbGl2ZQ%3d%3d#db=psyh&AN=2014-44350-008 I chose this article because it supported the finding that our perception of color influences our interpretation of events, specifically sporting events in this study. It also supports the idea that we innately associate some colors with power and success, and others with weakness inferiority.

http://web.b.ebscohost.com.proxy.lib.uni.edu/ehost/detail/detail?vid=15&sid=b3320392-0d0b-4d0a-ba84-f7f9b423af5a%40sessionmgr110&hid=109&bdata=JnNpdGU9ZWhvc3QtbGl2ZQ%3d%3d#db=psyh&AN=2014-17814-001 I chose this article because it found strong evidence for color perception being an important part of our daily social interactions by allowing us to determine the emotional and physical state of a person.

The topic I chose to discuss was color blindness.

Color blindness relates to the chapter because vision is a large part of sensation and perception. What we see or what we think we see impacts our views and everyday lives. Not everyone has the same visual abilities and chapter 5 discusses many of the problems people may have with seeing colors and how the rods and cones affect our eyesight.

I am interested in this topic because I have always been fascinated in the abnormalities and disorders people can acquire. Learning about the causes and effects and other aspects of each disorder makes me understand better the people who do not suffer these disorders. I like knowing what is normal but I also like knowing what isn’t necessarily normal in people.

The human eye can see light when the retina is stimulated. Rods and cones are what the retina is made up of. The rods are located in the peripheral retina and these are what give us our night vision but are not what distinguishes the color. Cones are located in the macula which is in the center of the retina and aren’t very helpful at night but do let us perceive color in the daylight. These cones contain light sensitive pigment which is sensitive over a range of wavelengths according to http://colorvisiontesting.com/color2.htm. Each visible color is a different wavelength and genes play a role in producing these coding instructions to develop these pigments. If the coding instructions are wrong then the wrong pigments will be produced and this can result in color deficiency.

When you hear someone say that they are color blind or they know someone who is color blind, most people think they these people can only see in black and white. This actually is not the case. It actually is very, very rare to see in complete black and white and be completely color blind. The complete absence of color is called monochromasy. There are different kinds of color blindness and it is actually referred to as color vision deficiencies. People with normal cones and light sensitive pigment have the ability to see all of the different colors using cones sensitive to one of the three wavelength of light – red, green, and blue. (http://colorvisiontesting.com/color2.htm) A mild color deficiency is present when one or more of the three cones light sensitive pigments when the development wasn’t right or it was shifted. Statistics show that 5% to 8% of men and .5% of women in this world are born with some form of colorblindness.

Deutan color vision deficiencies are the most common forms of color blindness. Statistics show that about 6% of the male population has this form of color blindness. A deuteranopic person has trouble seeing reds and greens. In monochromatism, one either has one type of cone present or none at all. In dichromatism, one either has only two different types of cones and the third one is missing completely. A person with anomalous trichomatism one has all three types of cones but they are just shifted and the sensitivity is affected. Protanopia is the disorder where one cannot see long wavelength red light. There is a difference in some of these types of color blindness. There are differences between red color blindness and green color blindness. Red-blind people perceive the color red much darker than what it actually is.

Some people say that the difference between people who can see color verses people who have a disorder relating to color blindness is that “colorblind people see the world like people with normal color vision see it at dusk or dawn.” During dusk or dawn, people have trouble seeing colors clearly and this would be comparable to the lifestyle of a person who has a form of color blindness. People who suffer from a disorder related to color blindness eventually adjust to it and can still make interpretations based on what colors they can see.

Terms: Visual system, colorblindness, visual system, deutan, deuteranopic, monochromatism, dischromatism, anomalous trichomatism, protanopia, rods, cones, disorders, pigment, wavelength, color deficiency.

http://colorvisiontesting.com/color2.htm. – I liked this website because it gave me a lot of information about how color blindness is developed and the causes of it. I think it is important to know how a disorder is developed before getting to know more about the disorder itself.

http://www.color-blindness.com/2010/03/09/types-of-color-blindness/ - I chose to use this website because I ran into a lot of the same information as what was on the other websites so I also know this is a credible source. It went into more detail about the types of color blindness rather than just sticking to what color blindness is.

http://www.webmd.com/eye-health/tc/color-blindness-topic-overview - I used this website because first of all WebMD is a very highly recommended website by many people and the information on it is all accurate and it is a credible source. I liked how the site is always set up because it gives me information about everything associated with what I am looking for. It gives me information about symptoms, treatments, diagnosis, coping with a disorder

1a) State what your topic is: Achromatopsia

1b) Discuss how the topic relates to the chapter.

This is directly related to chapter 5 as it is explained on page 133 of our textbook. Achromatopsia is the inability to perceive colors that is caused by damage to the central nervous system.

1c) Discuss why you are interested in it.
Honestly, no one in my family has this, nor have I ever met anyone with this, so I was most intrigued by the subject and how a person is affected by not seeing color. Our world is filled with many ways we use color as a means of communication and I wondered how people were affected by not seeing color. I was also curious on whether or not this could be treated, corrected and if so, how could someone just live with never seeing color.

I also wanted to find out if this was hereditary.

After researching the subject of Achromatopsia I found that there are two types of achromatopsia: congenital achromatopsia and cerebral achromatopsia.
Achromatopsia (congenial) is a non-progressive and hereditary visual disorder which is characterized by decreased vision, light sensitivity, and the absence of color vision. People with complete achromatopsia cannot perceive any colors; they see only black, white, and shades of gray. (There is also incomplete achromatopsia which is a milder form of the condition that allows some color discrimination.)

Getting further into the subject of achromatopsia, it is also known as a disorder of the retina. The retina contains two types of light receptor cells, called rods and cones. These cells transmit visual signals from the eye to the brain through a process called phototransduction. Rods provide vision in low light (night vision). Cones provide vision in bright light (daylight vision), including color vision.

It is considered rare, as statistics show that one in every 33,000 - 40,000 people are affected. The numbers vary in different parts of the world. From my research, due to the genetic link, it is more common in regions where there is a high rate of consanguineous marriages (marriages between relatives), the eastern Pacific islands, the Pingelapese islanders (they live on one of the Eastern Caroline Islands of Micronesia). It is said that between 4 and 10 percent of people in this population have a total absence of color vision. (WOW!)

Another fact I found during my research is that it happens right at conception. Each sibling of an affected individual has a 25% chance of being affected, a 50% chance of being an asymptomatic carrier, and a 25% chance of being unaffected and not a carrier. Carrier testing for at-risk family members and prenatal testing for pregnancies at increased risk are possible once the disease-causing mutations have been identified in the family.

The second type of achromatopsia which should never be confused with congential is cerebral achromatopsia, also known as the acquired form of total colorblindness that can result from trauma, illness, or some other cause. People who develop cerebral achromatopsia report that they see a monochromatic world, all in shades of gray. They are able to see gray because they previously experienced color vision, making it possible for them to perceive the absence of color as gray. This is in sharp contrast to the visual perception of congenital, complete achromats (complete rod monochromats), who report that the concept of "gray" is as mystifying to them as is the concept of any of the other colors. People with cerebral achromatopsia are diagnosed by neurologists, rather than eye specialists. Their loss of color perception is not accompanied by severely impaired vision, extreme light sensitivity, or abnormality in the photoreceptors of the retina, as is the case with people who have congenital, inherited achromatopsia.

Currently there is no cure for achromatopsia. But there are studies and some treatments to help live with achromatopsia.

The National Eye Institute currently has a study going on involving a CNTF implant. According to the research, CNTF is a natural chemical found in the body that promotes the survival and functional a nerve cells. This implant’s hope is to learn whether CNTF can improve visual acuity or color vision, and whether it can reduce sensitivity to light.

There are also dark or special filter glasses or red-tinted contact lenses to reduce photophobia and potentially improve visual acuity; low vision aids; and occupational aids.

http://www.achromat.org/ - I used this reference as a starting point for researching my topic/subject. This website is a network of information for individuals and families who are affected by achromatopsia. There is a pdf of a book called “UNDERSTANDING AND COPING WITH ACHROMATOPSIA” by Frances Futterman that I used as a reference when writing about my topic.

http://www.ncbi.nlm.nih.gov/pubmed/20301591 - I choose this website as a reference to the heredity question and also for the massive amount of information it contained on the subject of achromatopsia. This website is run by The National Center for Biotechnology Information and provides access to the biomedical and genomic parts of many subjects. The information taken from this website can be linked to the second paragraph.

https://clinicaltrials.gov/ct2/show/NCT01648452 - This website provided me with the information on one of the most current studies by the National Eye Institute (NEI) for treatment of achromatopsia. (The study is ongoing but not recruiting participants.) The data taken from this reference can be directed linked to the end of my blog.

Terms: Achromatopsia, congenial, central nervous system, vision, light sensitivity, studies, National Eye Institute (NEI), data, National Center for Biotechnology Information, biomedical, genomic, subjects, treatment, color, understanding and coping, visual disorder, color sensitivity, cerebral achromatopsia, colorblind, rods, cones, blind, eye, specialists, diagnosed, monochromatic, contrast, visual perception, rods, photoreceptors, neurologists, retina, glasses, contacts, asymptomatic, and chemicals.

This week for my topical blog I choose to look further into color blindness. This relates back to the chapter as it talks briefly about color blindness, but looking further into the visual system. The chapter talked a lot about how the eyes perceive color but more into the detail of the different cones and wavelengths. I thought that this would be a interesting topic to look further into because it is hard for me to imagine a life with no color. Color is something that we see everyday and to just wake up and no longer see color or imagine a life where you never have seen color is hard for me to understand.
Color blindness is not actually being blind or any form of blindness, but in fact just a deficiency in the way you see color.The Human eye sees light by stimulating the retina which is made up of rods and cones. Rods are used at night but do not distinguish color. Cones are not good at night but do let us perceive color during the daylight condition. Often times many people think that if a person is colorblind that the only thing they see is black and white which is in most cases not true. Many people with color blindness have difficulty distinguishing certain colors such as red or green. Studies have shown that color blindness is an inherited condition that affects males more frequently than females. With the most common form of color blindness being red-green.
There are different symptoms that vary from person to person but may include trouble seeing colors and the brightness of colors in the usual way. Inability to tell the difference between shades of the same or similar colors. Often times symptoms may be so mild that people may not even know that they are color blind. Rapid side to side eye movements and other symptoms may occur in severe cases.
There are many different reasons as to why someone may develop color blindness but one way is through inheriting it from someone else in your family. Besides different genetic makeup there are other causes of color vision defects of loss like parkinson’s disease, cataracts, tiagabine for epilepsy, Leber’s hereditary optic neuropathy and kallmann's syndrome. Color blindness can also occur when aging processes damage retinal cells.
There is no cure for color blindness but there are coping strategies that may help one function better in a color-oriented world. Most people are able to adapt to color blindness without much troubles but some professions like graphic design may suffer. It is important to work with the eye doctor if you feel that you have occurrence of color blindness and can learn more about treatment options that are right for each and every person.

Findings:
http://www.allaboutvision.com/conditions/colordeficiency.htm- this website was very informational and had alot of information in regards to what someone might feel and how one would react from the situation. It was very knowledgeable but also “dumbed” down at the same time.

http://colorvisiontesting.com/color2.htm- this one was harder to understand but talked a lot about the gender differences and how females developed the color blindness less often than men.

http://www.ncbi.nlm.nih.gov/pubmedhealth/PMH0001997/- this website was good to start with to give simple and general knowlege about the topic before broading out to other websites to search and was very good for doing the main defenition.

Terms: color blindness, wavelengths. genes. cones. rods.parkinsons disease, cataracts, tiagabine for epilepsy,

My topic is achromatopsia. This is related to the chapter because it is a rare subtype of color blindness. I am interested in this because I really love colors. The thought of not seeing any colors, like in this type of colorblindness, is very sad to me. Therefore, I wanted to find out the exact mechanics behind it. I also wanted to learn about the effects of the condition on the actual people who are afflicted.

Achromatopsia is a rare disorder that occurs in 1 of every 33,000 people in the United States. It is a type of colorblindness characterized by the presence of malfunctioned cones in the retina. Cones are a type a photoreceptor used to detect colors in daytime. In achromatopsia, the cones do not work correctly, which only allows vision in black, white, and gray. There is another type of achromatopsia called incomplete achromatopsia or blue cone monochromatism. This is when only the S-cones function properly. This leaves only cones that pick up shades of blue and rods, the nighttime photoreceptor to see with. This type is even rarer than complete achromatopsia, and predominately occurs in men at anywhere between 1 in 50,000 and 1 in 100,000. The odds of a female getting this disorder are nearly 1 in 10 billion.
Not being able to see color is much more debilitating than one might think. Due to the fact that those with achromatopsia cannot use their cones, they must rely on their rods in order to see. This is a considerable problem because rods are meant for vision in low light. This leads to hemeralopa, or day blindness. Day blindness refers to when what one sees appears to extremely bright. Those with day blindness are driven to squint in all non low-light situations. This is closely linked with photophobia, which is light sensitivity. Another side effect of achromatopsia is pendular nystagmus. This is a term for uncontrollable quivering of the eye. This disorder also causes greatly reduced visual acuity. This in turn can lead to problems and difficulty in school and at work.
The side effects of achromatiopsia can be especially difficult for children. If a child squints constantly from the hemeralopa and has jerky eye movements from nystagmus, they could face ridicule in school. Luckily there are ways to alleviate the negative effects. For example, there are special red or amber colored glasses and contacts that can be used to correct the hemeralopa and possibly even aid in visual acuity. As for nystagmus, this usually only occurs in young children and eventualy tapers off as a person grows older. This goes to show that even though achromatopsia is a troubling disorder, there are still many ways to improve quality of life.

Terms: achromatopsia; colorblindness; cones; retina; photoreceptors; S-cone; Blue cone monochromatism; rods; hemeralopa; day blindness; nystagmus

Sources:
http://www.aapos.org/terms/conditions/10
I liked this site because it provided clear and concise basics of the disorder. It acted as a basis for me to understand everything. I trust it because it is an official association's webpage.

http://www.achromatopsia.info
I liked this site because it was a webpage for people with achromatopsia. This gave it a more personal view with a lot of good information and videos to help explain concepts. The videos helped me to see this in the lives of actual people.

http://www.ncbi.nlm.nih.gov/books/NBK1418/
This site was helpful because it was so technical and in depth. It allowed me to find medically accurate terms and definitions for the topic areas.

1. A) My topic is achromatopsia.
1. B) This subject is related to the chapter because it is a rare subtype of color blindness, and the chapter was about the perception of color. I am interested in this disorder because I really love colors. The thought of not seeing any colors, like in this type of colorblindness, is very depressing to me. Therefore, I wanted to find out the exact mechanics behind it. I also wanted to learn about any effects of the condition on the actual people who are afflicted, besides the obvious.

2.) Achromatopsia is a rare disorder that affects one’s color vision. It typically occurs in 1 of every 33,000 people in the United States. Achromatopsia is a type of colorblindness. It is characterized by the presence of malfunctioned cones within the retina. These cones are a type a photoreceptor that are used to detect colors in the daytime or under lighted conditions. In complete achromatopsia, none of the cones are able to work correctly. This, in turn, only allows for vision in black, white, and gray. There is also another, less dramatic type of achromatopsia. This is called incomplete achromatopsia or blue cone monochromatism. This occurs when only one type of cones, the S-cones, function properly. Since the S-cones perceive the wavelengths of light that produce shades of blue, those with this disorder can only see shades of blue in the day time. This type is even rarer than complete achromatopsia, and predominately occurs in men at anywhere between 1 in 50,000 and 1 in 100,000. The odds of a female getting this disorder are nearly 1 in 10 billion. In both types of achromatopsia, the rods, or nighttime photorecptors are not affected. This means that vision in low lights situations is little changed.

Not being able to see color is much more debilitating than one might initially think. Due to the fact that those with achromatopsia are unable to use their cones, they must rely on their rods in order to see. This is a considerable problem because rods, as mentioned previously, are meant for vision in low light situations. When a person is forced to use their rods for daytime vision, it can lead to hemeralopa. This is also known as day blindness. Day blindness is the term that describes a type of “blindness” caused by exceeding brightness perceived by the eyes. This can be much like walking out of a dark theatre into the bright sun, but the eyes of individuals with hemeralopa do not ever “adjust to the light.” Because of this, these people are driven to squint constantly in all non low-light situations, effectively narrowing their field of vision. This problem is very closely linked with photophobia, which is light sensitivity. Obviously, this is quite a harrowing side effect. Another side effect of achromatopsia is something called pendular nystagmus. This is a term for an uncontrollable quivering of the eye. Pendualr nystagmus leads to greatly reduced visual acuity. As with most difficulties in vision acuity, this will most likely lead to problems and difficulty in school and at work.

The side effects of achromatiopsia can be very difficult. This goes even moreso for children. If a child is driven to squint constantly because of their hemeralopa, or perhaps has jerky eye movements due to their pendular nystagmus, they could face harsh ridicule in school. As we all know well, bullying in school is very difficult to endure. Luckily there are ways to alleviate the negative effects. For example, there are special red or amber colored glasses and contacts that can be used to correct the hemeralopa and possibly even aid in visual acuity. These special lenses act like sunglasses and reduce the impact of the light on the eyes. As for pendular nystagmus, this usually only occurs in young children. The condition will most likely eventually taper off as a person grows older. These positives go to show that even though achromatopsia is a troubling disorder, there are still many ways to improve quality of life.

Terms: achromatopsia; perception; colorblindness; cones; retina; photoreceptors; S-cone; Blue cone monochromatism; rods; hemeralopa; day blindness; pendular nystagmus; visual acuity

Sources:
http://www.aapos.org/terms/conditions/10
I liked this site because it provided clear and concise basics of the disorder. It acted as a basis for me to understand everything. I trust it because it is an official association's webpage.

http://www.achromatopsia.info
I liked this site because it was a webpage for people with achromatopsia. This gave it a more personal view with a lot of good information and videos to help explain concepts. The videos helped me to see this in the lives of actual people.

http://www.ncbi.nlm.nih.gov/books/NBK1418/
This site was helpful because it was so technical and in depth. It allowed me to find medically accurate terms and definitions for the topic areas.

1a) Agnosia

1b) Although this disorder relates more to specific objects than colors, this topic does relate to chapter five because it discussed how one might be able to see colors but not recognize the object that s/he was observing.

1c) I chose this topic because I wanted to learn more about it. Agnosia seemed so strange to me because I simply don’t understand how someone can see an object but not recognize it.

2) Visual agnosia is characterized by the inability to recognize familiar objects and/or faces without losing the ability to actually see them. This means that they are able to see the object or face in front of them, but have no or little idea what they are looking at. Take Kevin Chappell. He suffers from Visual Agnosia and has lost the ability to recognize objects which, in turn, has made it impossible to read words or music. However, Chappell can still recognize faces. He explains that the reason that he can still recognize a familiar face is because the ability to distinguish different objects and the ability to identify different faces come from separate parts of the brain. This would be Prosopagnosia.

An important thing that I found when dealing with understanding Visual Agnosia is that is brain damage that impairs the vision but not intelligence, attention, or motivation. Just because someone suffers a brain injury and it results in Agnosia does not make them intellectually impaired as well. Back to Kevin Chappell, he was seen playing the guitar flawlessly throughout the entirety of the video. Some patients have a form of primary Visual Agnosia that makes their surroundings unfamiliar as well.

Different things can cause Agnosia, but they can all be traced back to brain damage. Things like strokes, overexposure to dangerous toxins, or, in Kevin Chappell’s case, a car accident resulting in brain injury are all ways that a disorder like this can occur.

Something that I found interesting in my search was the tie to Alzheimer’s disease. This disease affects the brain’s ability to recognize visual input. Because of this, Agnosia can result in the form of the inability to recognize objects, like the type that has been discussed, or prosopagnosia, the inability to recognize faces.

3) Key Terms: Agnosia, disorder, colors, recognize, ability, prosopagnosia, brain damage, intelligence, Alzheimer’s disease, input,

4) https://www.youtube.com/watch?v=ze8VVtBgK7A

I chose this video because it gave a good basis and real life example of the disorder.

http://www.rarediseases.org/rare-disease-information/rare-diseases/byID/984/printFullReport

This website provided a good information source for the details of the disorder.

http://www.dementiaguide.com/aboutdementia/alzheimers/agnosia/

This website helped with the tie between Agnosia and Alzheimer’s.

The study of color vision is especially interesting given the nocturnal habits of animals. In chapter 5, we learn that cone photoreceptors are divided into type, depending on what wavelength they are specialized for (S, M, or L). As humans and many types of primates rely more on vision than other senses, it is particularly interesting to study the effects of evolution and nocturnal activity on their photoreceptors and visual interpretation. The Bayesian approach uses a working knowledge of the world to unconsciously assign probability to various scenarios which might serve as explanations for a given scene. We then use a committee-based decision making strategy to figure out what we are looking at (all of which occurs without a conscious thought). The idea that vision relies on implicit understanding and photoreceptor stimulation indicates that it is an evolutionary trait, rather than a learned activity. With this in mind, we can explore the role of evolution in color vision.
In nocturnal animals, color vision plays an important role in survival. The folivorous wooly lemur is a nocturnal member of the primate family, making it a good study for this research question. This specific type of lemur seems to have developed photoreceptors especially responsive to certain green leaves that they prefer eating. Without this adaptation, it is unclear if they could identify primary food sources. Additionally, it is not likely other species would benefit from the same adaptation, unless they favor the same green leaves.
Quite a bit of research has explored the color vision of marsupials, a special case which evolved separately from humans or primates. In particular, the tammar wallaby is known to have dichromatic vision, and is in fact the only species to show no evidence of a third cone photoreceptor. Dichromatic vision is a condition where only two types of cone photoreceptors are present, instead of the three that we have. As a group, marsupials are largely thought to be dichromats, although ongoing research may be able to confirm using behavioral research methods.
With more research, we’ll know more details about the various differences in color vision among species. In some cases, studies have shown how certain species or groups have adapted their color vision for a better chance of survival. For now, we can be sure evolution will continue to take its course in prompting these helpful adaptations. Taken as a whole, photoreceptors are highly specialized for detecting certain types of light, including certain ranges of wavelength that make up the color spectrum. Evolution has already given many examples of how photoreceptors can be highly specialized to benefit the organism.

Terms: Color vision, cone photoreceptors, wavelength, Bayesian approach, dichromat,

http://www.ncbi.nlm.nih.gov/pubmed/18627773
Color vision in humans and animals is extremely varied depending on the needs of the animal for cone and rod photoreceptors.

http://eds.b.ebscohost.com.proxy.lib.uni.edu/eds/pdfviewer/pdfviewer?vid=2&sid=9680c1d5-5eab-4464-8658-73554793a3ba@sessionmgr111&hid=104
This article gave some good insights on testing color vision, particularly of marsupials. The tammar wallaby is a special case study highlighting the behavioral approach to studying color vision.

http://eds.b.ebscohost.com.proxy.lib.uni.edu/eds/pdfviewer/pdfviewer?vid=2&sid=63a696f8-1c7f-4c65-906f-23b938451cc2@sessionmgr113&hid=104
This article explores the color vision of the folivorous wooly lemur and how their photoreceptors have adapted for nocturnal activities.

Color blindness, or color vision deficiency as it should be called, is present in about 8% of males and less than 1% of females. The most common form of color vision deficiency is red-green. The trait for blue-yellow vision deficiency is even more rare than that of red-green and affects women and men equally. Most people who are color blind can still see colors, it is just that some seem washed out depending on the deficiency and severity, it is extremely rare to have someone who only sees shades of grey. In the eye there are three different kinds of cones, ones that respond to red, blue, and green light. An inherited deficiency in either of these 3 cone types can cause color blindness. Why are men more affected on average than women? Because the genes for color blindness are located on the X chromosome, and in order for a woman to be affected by that gene she would need the defect to be present on both X chromosomes, whereas where men only have one X chromosome, if it carries the gene they will have color vision deficiency.

https://www.youtube.com/watch?v=-Iz8xZD9LcI

The topic I chose this week was the trichromatic theory of color vision. I chose this because I really didn’t completely understand it in the text. I also love colors and it fascinated me that all the various hues three cones or receptor types and that makes up the thousands of colors we see every day. There is so much in this class that we just simply daily see that in reality is the working of very intricate actions in the brain and eye.

This theory says that there are three cones that perceive the colors. These are the S, M and L cones. Short, medium and long, each responds to a color. So since a blue has a short wave length the S cone is activated, the color green has medium wave length so the M cone is sensitive to it and red is long wave and the L cone is sensitive to it. As the colors come at us the impulses trigger the cones each color reacts within the specific cones and we see the color. What I found really interesting was that the video showed shining the color yellow through a prism it is still seen as yellow but a mixture of red and green is perceived as a combination with yellow at the center.

There is more to seeing the color than simply the three cones. The cones are connected to other nerve cells. A ganglion cell gathers the information from the nerves and gives the response of the color for a particular area of the retina. The ganglion has a center and a surrounding area and each are fed by different types of cones. It kind of reminded me of the binary 0,1. In this case it is + if the center is excited by the stimulus and – in the surrounding area. The surround area is called an off because it inhibits not stimulates. This can be flipped and the center can be off and surround be on. What seemed to be was that this is only part of the perception of color. There is more to our sensing of color than the three cones.

One thing I really get is how more can be added but it doesn’t necessarily have to debunk earlier findings. There was quite a bit I really struggled with understanding, so hopefully I can ask rather than answer questions on Thursday. It is also why this post is rather short in comparison with my other posts. I struggled with finding very much on the internet about the subject that wasn’t either a paragraph long or else written way over my head. My best information came from the videos I watched.

Terms: trichromatic theory, color vision, cones, receptors, retina, ganglion, nerve, stimulus


https://www.youtube.com/watch?v=V73k_0KuUJo The video gave a lot of good information and watching it was easier than reading it.

http://en.wikipedia.org/wiki/Young%E2%80%93Helmholtz_theory

http://webvision.med.utah.edu/book/part-viii-gabac-receptors/color-perception/
The two web pages has some good history on the theory, as well as some good information, but not as easy to get through as the video

1a) My topics for this week are colorblindness and EnChroma glasses

1b) Colorblindness was brought up several times in this last chapter and because of that I wanted to expand on the subject even more.

1c) I am interested in this topic because I have friends that are colorblind, and I was interested in the new technologies out there that could help with fixing the problem. Since I was little I have always found it interesting how my colorblind friends perceive things in the world.

2) Colorblindness is more common in men than women, because the genes that code for the M and L cone deficiencies are located on the X chromosome which men only have one of. For women they need the deficiency on both of their X chromosomes to have these types of colorblindness. The third type of colorblindness is with S cones which is located elsewhere on the DNA code making it the rarest form. Even with colorblindness, people still are able to see items in the world clearly based on the different wavelengths that are being receipted in the cones that they do have. The deficiencies vary from case to case; sometimes it is very small and unnoticeable. In these cases the individual may not even know they have a deficiency. If they have a high deficiency then they would not be able to see certain colors. One interesting fact found through experiment was that females with colorblindness female carriers were found to have more difficulty in perceiving patterns of colored stimuli than men with no colorblindness.

The three types of Colorblindness are deuteranope, protanope, and tritanope, each based on a different cone deficiency (L, M, S cones). We also use the term color-anomalous to refer to “color-blind.” Most people that are colorblind can still differentiate between shades based on wavelength.

With new research coming out every year, so are new technologies. In recent news there has been a discovery by filtering the wavelengths of light by way of sunglasses. By doing this the color signal sent to the brain can be amplified. They coat the glasses with layers of dielectric material which makes photons passing through the coatings selectively blocked depending on their wavelength. One kind of these glasses are called EnChroma Cx which helps with red green deficiencies from deuteranope and sometimes proanope.

Terms: Colorblindness, deuteranope, protanope, tritanope, wavelength, cones, color-anomalous

http://enchroma.com/technology/how-it-works/ The is the main website for the glasses that correct red green colorblindness, it also describes how it works and why.

http://web.a.ebscohost.com.proxy.lib.uni.edu/ehost/detail/detail?vid=4&sid=830faf61-02d5-4f6e-b0ba-fd4773c86e5b%40sessionmgr4004&hid=4104&bdata=JnNpdGU9ZWhvc3QtbGl2ZQ%3d%3d#db=psyh&AN=1989-29892-001 This was an experiment done with colorblind females.

http://web.a.ebscohost.com.proxy.lib.uni.edu/ehost/detail/detail?vid=5&sid=830faf61-02d5-4f6e-b0ba-fd4773c86e5b%40sessionmgr4004&hid=4104&bdata=JnNpdGU9ZWhvc3QtbGl2ZQ%3d%3d#db=psyh&AN=2005-12693-004 This is a review of an originally German written document on colorblindness, I mostly choose this rather than the actually article because Mien Deutch ist nicht gut. (My German is not good)

I decided to research the topic of colorblindness. This relates to the chapter, because it was mentioned in the chapter and it concerns our ability to process the information that we are receiving from the outside world. I chose this topic, because I know several people who have difficulty with many daily tasks because of their color blindness.

Color blindness is the inability or decreased ability to see color or to perceive color differences under normal lighting conditions. The most common cause of this is a developmental problem with or ore more sets of cones in the retina that perceive color and then transmit the information to the optic nerve. This cause is genetic and affects mostly males. Color blindness can also be caused by Parkinson's disease, cataracts, Tiagabine (a drug used to treat epilepsy), Leber's hereditary optic neuropathy, Kallman's syndrome, diabetes, aging, or physical damage to the eye.
There are three main types of color blindness: monochromacy, dichromacy, and anomalous trichromacy. There are also several subtypes within these main types. The most severe type is Monochramacy. This is the condition of possessing only a single channel for conveying information about color. The technical term for this is achromatopsia, or total color blindness, which is the complete inability to see color. The most common type of color blindness is red-green color blindness. This when one is unable to or has difficulty distinguishing red and green hues because of the absence or mutation of the red or green retinal photoreceptors. This type is part of the dichromacy color blindness. This when someone can match any color they see with some mixture of just two primary colors. This is different from people with fully functioning color perception that require all three primary colors. Because of this, certain pairs of colors, like red and green, that seem quite different to the normal viewer might appear to be the same color to someone with this type of color blindness. Dichromats often have difficulty with daily tasks like picking out clothes to wear or distinguishing between the colors of a stoplight. The final type is anomalous trichromacy. People with this type require different color matches from people with normal vision. For example, in order to match a given spectral yellow light, they may need more red light or green light in a red/green mixture than a normal viewer.
Color blindness has been cured in monkeys using gene therapy, but this will not be available to humans until it can be proven to be safe. The only other treatment available for color blindness is filtered lenses that enhance color perception. These lenses can be difficult to find, though, as they are only available from a limited number of eye care practitioners. It is mostly up to the affected person to adjust their lives accordingly with the condition. One may need to memorize the order of stop lights rather than the colors or organize their clothes in a way that will prevent them from accidentally put on clashing colors.

Terms: Color blindness, Cones, Achromatopsia, Photoreceptors, Monochromacy, Dichromacy, Anomalous Trichromacy, Perception

https://www.youtube.com/watch?v=8Aaivktz8G0 I used this source, because it had all of the basic background information on the topic and presented it in an easy to process manner.

http://en.wikipedia.org/wiki/Color_blindness I used this source, because it had a lot of technical information that went further into depth than many of the other sources I found.

http://www.allaboutvision.com/conditions/colordeficiency.htm I used this source, because it focused more on how people with color blindness are affected by the condition, rather than just the technical information about the condition itself.

1. The topic I picked to explore is human night vision. This topic is related to the chapter because chapter 5 talked about night vision. I wanted to learn more about why people have worse night vision when compared to other animals and if it’s possible to not be able to see in the dark at all.
2. Basically humans terrible eye sight when compared to other animals because humans have fewer cones than nocturnal animals and the cones that we do have are less sensitive. The cones we do have work with the rods all the time (aka the Dual Receptor System), but cones are especially active when trying to see in low light. There are two types of Ranges that impact species night vision ability: Spectral Range and Intensity range. Spectral range is the radiation level visible (infrared or ultraviolet). Humans are not able to use as much of the spectral range as other animals. Snakes are example of animals that use thermal vision to the extent they could go for a mouse in pitch black and still catch it. Intensity Range is the ability to see in very low light. Humans are not able to do this very well. For example, an owl could see a mouse on a football field if there was only one candle burning in the field.
Humans are also incapable of seeing most of the spectrum of light, as we are stuck with only the visible light portion. This explains why everything we see while in low light is dimmer, as the cones do not pick out most colors. The only color our eyes see in shades of black, white and gray, a moonlit night is an example of this. This could also be described as Dark Adaptation. This type of adaptation works because the photo pigments analyses the amount of light available for the cones in a process called light adaption (where light energy is converted to electrical impulses). Cones do a better job of this process because it only takes 5-7 minutes while it take the rods 30-45 minutes to do the same thing: make sure the eye is as prepared for light as it can be.
During my research, I also discovered a few things about night vision I did not know before I started this assignment. The first thing I found surprising is that a human’s peripheral vision is better in dim light than bright light. This is because there are more light sensitive rods towards the back of the eye. This increases the acuity of the peripheral vision. This can create a central blind spot because the fovea cannot focus at light levels lower than Dim Starlight. The other thing I found to be interesting was how a person’s myopia (nearsightedness) can get worse or cause myopia to show as a problem. This occurs to flyers because of the reduction of light causes issues with how effectively the fovea focuses. The extreme form of this problem is called night blindness. Night blindness can be extreme enough to stop people from driving at night. Night blindness has a variety of causes: cataracts, myopia, drug use, vitamin A deficiency, birth defects, and Retinitis Pigmentosa (damaged retina). Night blindness only has two untreatable causes: Birth defects and Reginitis Pigmentosa.
No matter how good someone’s night vision is: there are a few devices that can be used to improve it. Image intensification works by taking natural sources of light and magnifying them for use in the space surrounding them. A flashlight is an example of how this works. Active illumination is when the device being used is able to use near infrared light that the human eye is not able to see. CCD cameras (security cameras) are example of this concept. Thermal vision works by measuring heat levels in the room and using those to levels to figure out what is in foreground and background. The way snake hunt is an example of this.
One animal that won’t need any of these devices I just described is the cat. They have the best night vision of all mammals because they have a wider field of view than humans and the differences in their eyes. Cats have a 200 degree field of vision, while humans only have 180 degrees. Cats eyes also have Elliptical shaped corneas that are larger than humans as anyone who has ever seen a cat who’s pupils seemed to range between elliptical and so big seems they have black eyes. Cats also have a Tapetum (tissue layer that reflects light back into retina which creates the glow-in-the-dark look to their eyes at low lights) On top of having 6 or so more rods than humans. These traits are important as cats are most lively at dawn and dusk which makes them crepuscular on top of being nocturnal. Despite all these cool traits, cats do have more trouble with motion detection in bright light and are thought to have color vision similar to dogs.
Terms: night vision, cones, nocturnal animals , sensitive,Dual Receptor System, rods, low light, Ranges, Spectral Range and Intensity range, infrared, ultraviolet, thermal vision , spectrum of ligh, visible light , dimmer, black, white and gray, Dark Adaptation, electrical impulses, light energy, converted, photopigments , light adaption ,peripheral vision, light sensitive rods , central blind spot , fovea, focus, Dim Starlight, myopia , nearsightedness, night blindness, cataracts, vitamin A deficiency, birth defects, Retinitis Pigmentosa , Image intensification, magnifying , Active illumination , CCD cameras , foreground ,background, near infrared , field of view , Elliptical shaped corneas , pupils, Tapetum, tissue layer , , retina, glow-in-the-dark , crepuscular, motion detection .
http://en.wikipedia.org/wiki/Night_vision --> I picked this website because it provided good background information and gave me some point to explore further. I used it for background information, different ranges, and night vision devices.
http://indianapublicmedia.org/amomentofscience/night-vision-humans-color/  I picked this website because it boils the basic information down it the most interesting topics. I used it for information on the colors seen at night and the bit about periapical vision.
http://www.aoa.org/optometrists/tools-and-resources/clinical-care-publications/aviation-vision/the-eye-and-night-vision?sso=y  I picked this site because it had new information on it. I used it for information on: dark adaption, mesopic vision, dark adaption times for cones and rods, central blind spot and night myopia.
http://largestfastestsmartest.co.uk/animals-with-the-best-vision-in-the-world/ I picked this site because it had interesting information and strengths of animals. I used it for information on snakes, owls and cats.
http://www.nlm.nih.gov/medlineplus/ency/article/003039.htm I picked this site because it had basic information on night blindness. I use the information on night blindness.
http://www.livescience.com/40459-what-do-cats-see.html I picked this site because it had interesting information about cat’s eyes. I used it for the last paragraph.

For this weeks research assignment, I chose to do more research on Achromatopsia. Achromatopsia is defined as a non-progressive and hereditary visual disorder which is characterized by decreased vision, light sensitivity, and the absence of color vision. Achromatopsia is sometimes called ‘Day Blindness’, as these subjects see better in subdued light. Persons with complete Achromatopsia will have reduced vision (20/200 or less) due to an abnormality of the retina. They also have no color vision, sensitivity to light (photophobia) and the presence of nystagmus (shaking of the eyes). This condition affects approximately one in 40,000 births. Its prevalence varies in different parts of the world. Because there is a genetic link, it is more common in regions where there is a high rate of consanguineous marriages (marriages between relatives) and in the eastern Pacific islands of Pingelap.

This hereditary visual disorder coincides with this chapter because this chapter starts to discuss our visual system in accordance to color. This chapter discusses which parts of the eye are susceptible to "seeing" colors and how we process that information.

I was interested in this topic because it is one of those abnormalities that I tend to focus on. The way I look at it, if we can understand 'what is wrong' maybe that will give us a better understanding of our visual system as a whole. Achromatopsia is caused by an abnormality of the retina, that portion of the eye responsible for “making the picture”. In the retina, there are three types of cells called cones that are responsible for normal color vision. These are the red, green, and blue cones. A balanced distribution of these cells is necessary for normal color vision. Sometimes people have a reduced complement of the cones, in which case they will have partial or incomplete achromatopsia. Since achromatopsia is an inherited condition and so far four genes are known to be associated with this condition. The four chromosomes that may have changes associated with achromatopsia are chromosome 14, chromosome 8q21- q22, chromosome 2q11 and chromosome 10q24. As I was reading through some of the research for this assignment, I came across studies that were being done across the nation. These studies were examining treatment plans and executions for dogs and mice. Research continues on developing genetic treatments for achromatopsia. In animal models of achromatopsia (dogs and mice), genetic therapy has shown positive results in restoring some cone function. Because the incidence of achromatopsia is rare compared to other retinal disorders, the amount of research and funding may be limited. However, the knowledge being gained from the more extensive genetic treatment research being done to treat conditions such as retinitis pigmentosa and macular degeneration, will likely advance the ability to genetically treat achromatopsia.

Currently there is no treatment for Achromatopsia, although deep red tinted glasses or contact lenses can reduce symptoms of light sensitivity. The potential treatment being tested uses an engineered adeno-associated virus (AAV), a safe, man-made virus that delivers healthy copies of the ACHM gene to the cells of the retina, replacing the defective copies of the gene. A single treatment is expected to stop the disease for at least several years, perhaps a lifetime. The AAV delivery system is successfully being used in clinical trials of Leber Congenital Amaurosis gene therapy that have restored vision in more than 50 adults and children who were virtually blind. Previous research has shown promising signs of efficacy in dog and mouse models of ACHM.

References:
http://www.aapos.org/terms/conditions/10 I chose this website because it gave a general background of this hereditary disease. It gave some visual aid as to what someone with this disease would look like and also how typically these abnormalities are found.

http://www.achromatopsia.info/achromatopsia-genetic-treatmen/ I chose to use this website because of the background information leading up to the clinical trials on dogs from mice. It also had a neat video showing the effects of the therapy vs. no therapy.

http://www.agtc.com/news/detail/the-university-of-florida-and-agtc-secure-8.4m-grant I chose this website because it gave "hope" that there is more money being funded to created some type of solution to this disease.

TERMS: Achromatopsia, color vision, 'Day-Blindness', retinitis pigmentosa, macular degeneration, light sensitivity, ACHM gene, cones, gene therapy retina, nystagmus, photophobia, hereditary visual disorder

For this weeks research assignment, I chose to do more research on Achromatopsia. Achromatopsia is defined as a non-progressive and hereditary visual disorder which is characterized by decreased vision, light sensitivity, and the absence of color vision.

Achromatopsia is sometimes called ‘Day Blindness’, as these subjects see better in subdued light. Persons with complete Achromatopsia will have reduced vision (20/200 or less) due to an abnormality of the retina. They also have no color vision, sensitivity to light (photophobia) and the presence of nystagmus (shaking of the eyes). This condition affects approximately one in 40,000 births. Its prevalence varies in different parts of the world. Because there is a genetic link, it is more common in regions where there is a high rate of consanguineous marriages (marriages between relatives) and in the eastern Pacific islands of Pingelap.

This hereditary visual disorder coincides with this chapter because this chapter starts to discuss our visual system in accordance to color. This chapter discusses which parts of the eye are susceptible to "seeing" colors and how we process that information.
I was interested in this topic because it is one of those abnormalities that I tend to focus on. The way I look at it, if we can understand 'what is wrong' maybe that will give us a better understanding of our visual system as a whole. Achromatopsia is caused by an abnormality of the retina, that portion of the eye responsible for “making the picture”. In the retina, there are three types of cells called cones that are responsible for normal color vision. These are the red, green, and blue cones. A balanced distribution of these cells is necessary for normal color vision. Sometimes people have a reduced complement of the cones, in which case they will have partial or incomplete achromatopsia. Since achromatopsia is an inherited condition and so far four genes are known to be associated with this condition. The four chromosomes that may have changes associated with achromatopsia are chromosome 14, chromosome 8q21- q22, chromosome 2q11 and chromosome 10q24. As I was reading through some of the research for this assignment, I came across studies that were being done across the nation. These studies were examining treatment plans and executions for dogs and mice. Research continues on developing genetic treatments for achromatopsia. In animal models of achromatopsia (dogs and mice), genetic therapy has shown positive results in restoring some cone function. Because the incidence of achromatopsia is rare compared to other retinal disorders, the amount of research and funding may be limited. However, the knowledge being gained from the more extensive genetic treatment research being done to treat conditions such as retinitis pigmentosa and macular degeneration, will likely advance the ability to genetically treat achromatopsia.

Currently there is no treatment for Achromatopsia, although deep red tinted glasses or contact lenses can reduce symptoms of light sensitivity. The potential treatment being tested uses an engineered adeno-associated virus (AAV), a safe, man-made virus that delivers healthy copies of the ACHM gene to the cells of the retina, replacing the defective copies of the gene. A single treatment is expected to stop the disease for at least several years, perhaps a lifetime. The AAV delivery system is successfully being used in clinical trials of Leber Congenital Amaurosis gene therapy that have restored vision in more than 50 adults and children who were virtually blind. Previous research has shown promising signs of efficacy in dog and mouse models of ACHM.

References:
http://www.aapos.org/terms/conditions/10 I chose this website because it gave a general background of this hereditary disease. It gave some visual aid as to what someone with this disease would look like and also how typically these abnormalities are found.

http://www.achromatopsia.info/achromatopsia-genetic-treatmen/ I chose to use this website because of the background information leading up to the clinical trials on dogs from mice. It also had a neat video showing the effects of the therapy vs. no therapy.

http://www.agtc.com/news/detail/the-university-of-florida-and-agtc-secure-8.4m-grant I chose this website because it gave "hope" that there is more money being funded to created some type of solution to this disease.

TERMS: Achromatopsia, color vision, 'Day-Blindness', retinitis pigmentosa, macular degeneration, light sensitivity, ACHM gene, cones, gene therapy retina, nystagmus, photophobia, hereditary visual disorder,

My topic for this blog is colorblindness:

Colorblindness relates to this chapter because "seeing" has so much to do with sensation and perception; and the colors we see have everything to do with the sensation we get from what we see.

Color blindness is a vision defect wherein the eye perceives some colors differently than others. This condition may be hereditary or may be caused by a disease of the optic nerve or retina.

Color blindness can be classified as inherited, partial or complete. Inherited color blindness is most common in males, affecting both eyes but does not worsen over time, this basically means that once you have it, you have it forever and there is no cure. Partial color blindness affects some colors while complete color blindness, which is a rare condition, affects all your color vision. Most people affected by color blindness see red instead of green or blue instead of yellow. Color blindness is usually classified as a disability; however, in some situations color blind people may have advantages over people with normal color vision. There is some evidence that color blind individuals are better at penetrating color camouflage and at least one scientific study confirms this under controlled conditions. Seeing how someone with one of the diagnosis we learn about throughout these chapters having an "advantage" is always interesting and nice to hear. Most of the males in my family are color blind however, it doesn't seem to affect any of them in their every day lives.

One weird thing that I have always wondered about colors is "how do we know that the colors we see are the same as what other see?" if we are told that "red" is red our entire life we would refer to it as red but maybe someone else sees your red as their blue and they just call it red because that is what they have always seen as red. (I'm not sure if that made sense at all) but colors have always been interesting to me. It is intriguing to know that we can never know what other people see and if they perceive things the same way that we do.


I think the worst type of color blindess would be; Congenital colorblindness is the primary type since it is a sex-linked trait, inherited on the X chromosome. People who have congenital colorblindness have never experienced color and do not understand the concept of gray. Congenital colorblindness is much more common in males since males have one X and one Y chromosome and females have two X chromosomes. Females are typically carriers for the disease because only one of their chromosomes are infected. It is very rare that both X chromosomes in a sex-linked disease are mutated since both parents would have to have be infected and give the disease on both of the daugther's X chromosomes which is not as likely as a son getting the one X chromosome.

Colorblindness can be very server or more mild but either way it is something that once diagnosed the person with it must deal with it for the rest of their lives.

Websites I used:
http://psychology.wikia.com/wiki/Color_blindness: I like this website because it talks abou6t how some people with colorblindness are actually at an advantage compared to those who are not.

http://www.color-blindness.com/; this website had quizzes to discover if you were colorblind yourself and also gave examples of how people deal with colorblindness and things they can do to cope with or deal with it.

http://users.start.ca/users/joneil/colour2.htm; this website was for people who are NOT color blind to try to get them to understand what people who are color blind experience. This is cool to me and interesting because of course we would all love to know how other people see/interpret/perceive things.

Terms: Terms: Visual system, colorblindness, visual system, dischromatism, anomalous trichomatism, protanopia, rods, cones, disorders, pigment, wavelength, color deficiency, perceive, sensation.

In chapter 5 I liked learning about impairments to color vision, because I have known a couple of people who were color blind, so i am curious as to how differently they view the world around them. I think it is difficult to understand the situation they are in, unless we were able to experience it ourselves, saying that I also think it may not need understanding, maybe it is just not a big deal to them since they have viewed the world in that way all of their lives.
How different areas of our brain serve specific functions, and how a person would have to adapt to not having a previously possessed ability, like color vision, after suffering damage to that area of their brain was intresting. Achromatopsia is a congenital eye defect that results in severe color blindness, poor detail vision, and photophobia. Photophobia is sensitivity to light. This is a hereditary condition that occurs more often in men than women. It is not progressive, and will not lead to complete blindness. The two types of photoreceptor cells in the cornea, rods and cones, are responsible for different functions of vision in the human eye. Rods specialize in allowing us to see in scotopic, or low, light levels, while cones provide sharp vision, color and contrast discrimination. The three different types of cone cells are responsible for our color vision, and they are packed by the millions into the macula of the retina. The peripheral retina is made of rod cells, and these cells are more sensitive to light but cannot differentiate color. People with achromatopsia have defective cone cells, and must rely on their rod photoreceptors for vision.
There’s a variety in the severity of symptoms of achromatopsia, with the most severe being called complete rod monochromatism. With complete rod monochromatism, there is a complete lack of cone function. Individuals suffering from this variation of achromatopsia, are extremely sensitive to light, even in normally light indoor rooms. They also have symptoms of poor visual acuity and nystagmus, which is involuntary movement of the eyes. Some other less severe variations of the disorder are called incomplete rod monochromatism and blue cone monochromatism, depending on which cones are affected.
Achromatopsia is an inherited condition, requiring both parents to contribute a gene for the condition to occur, which is very rare in today’s world. There is one group that has been studied in the Western Pacific that has a high incidence of achromatopsia. Through genealogy, it was traced back to 1775 to one man who survived a typhoon that killed most of the island’s residents. This man had a mutation of the CNGB3 gene, which is essential in the eye’s photoreceptors and ultimately vision. He passed this gene on to descendants who have subsequently been affected by achromatopsia. Today, 6% of the population of the small island of Pingelap has the condition.
A new treatment for individuals with achromatopsia is the use of red central soft contact lenses. These contact lenses have a small red circle that when properly positioned looks like the pupil of the eye. On the eye they appear as dark circles looking just like the normal pupil of the eye. These red contact lenses not only reduce the light entering each eye, but allow primarily red light to enter the eye. Red light allows the remaining rods to function better, and in complete achromatopsia, where the patient sees no color, the person isn’t aware of the red hue. In the incomplete form of achromatopsia, patients have reported that the contacts enhance their ability to detect red stoplights and red brake lights.
Cool stuff. Great to read that scientists are making strides in the area, helping those with sight issues.

Sources:
http://www.visionrx.com/library/enc/enc_achromatopsia.asp
http://www.achromatopsia.info/
http://www.achromat.org/what_is_achromatopsia.html

Terms: achromatopsia, photophobia, scotopic, complete rod monochromatism, visual acuity, nystagmus, incomplete rod monochromatism, blue cone monochromatism, red central soft contact lenses, pupil, hue

color blindness.
im doing color blindness not because so many others have done it but because my dad is partially colored blind and also because I have always wondered what it would be like to never know colors.
color blindness is such a crazy thing that ones eyes have devloped without the tools to detect color. people can go color blind when the rods and cones in the eye in someway get damaged. color blindess can effect your daily life if your driving down the street and you dont happen to get to a stop light that has red green and yellow in a differnt order then what your used to you could go right through it and get in an accident. because color blindess is a diffency in a way ones sees color. one might not be able to see red and green and all they see is yellow and the website says. the last website i have listed is a webiste where you can go to test your colorblindness. this is pretty cool to find out where you stand with color blindness. many people dont even know they are color blind untill somone says somthign to them because they were born that way and think its 100% normal.

http://www.allaboutvision.com/conditions/colordeficiency.htm
http://www.colormatters.com/color-and-vision/what-is-color-blindness
http://colorvisiontesting.com/

Color blindness is a deficiency in the way a person sees color and is usually difficulty distinguishing between blue and yellow or red and green. It is an inherited condition affecting men more frequently than women – about 8% of men and 1% of women. Red-green deficiency is most common. Light-sensitive cells in the retina fail to respond to variations in wavelengths of light. This results from an absence of color-sensitive pigment in the cone cells of the retina, the nerve layer at the back of the eye.
Terms: inherited, red-green deficiency, light-sensitive cells, retina, wavelengths, nerve layer

I decided to research the topic of colorblindness. This relates to the chapter, because it was mentioned in the chapter and it concerns our ability to process the information that we are receiving from the outside world. I chose this topic, because I know several people who have difficulty with many daily tasks because of their color blindness.

Color blindness is the inability or decreased ability to see color or to perceive color differences under normal lighting conditions. The most common cause of this is a developmental problem with sets of cones in the retina that perceive color and then transmit the information to the optic nerve. This cause is genetic and affects mostly males. Inherited color blindness can be congenital (from birth), or it can commence in childhood or adulthood. Depending on the mutation, it can be stationary, that is, remain the same throughout a person's lifetime, or progressive. As progressive phenotypes involve deterioration of the retina and other parts of the eye, certain forms of color blindness can progress to legal blindness, i.e., an acuity of 6/60 or worse, and often leave a person with complete blindness. Color blindness can also be caused by Parkinson's disease, cataracts, Tiagabine (a drug used to treat epilepsy), Leber's hereditary optic neuropathy, Kallman's syndrome, diabetes, aging, or physical damage to the eye. It can also be caused by damage to the brain such as through shaken baby syndrome and accidents and other trauma which produce swelling of the brain in the occipital lobe. Exposure to ultraviolet light can also damage the retina severely enough to cause color blindness. Often times, these damages present themselves later on in life. This means that one may not go color blind right when the damage happens, but rather years later on. There is not much data on the subject, but it is hypothesized that a deficiency in vitamin A may also be a contributor in color blindness.

There are three main types of color blindness: monochromacy, dichromacy, and anomalous trichromacy. There are also several subtypes within these main types. The most severe type is Monochramacy. This is the condition of possessing only a single channel for conveying information about color. The technical term for this is achromatopsia, or total color blindness, which is the complete inability to see color. Although the term may refer to acquired disorde such as cerebral achromatopsia, also known as color agnosia, it typically refers to congenital color vision disorders (more frequently rod monochromacy and less frequently cone monochromacy). In cerebral achromatopsia, a person cannot perceive colors even though the eyes are capable of distinguishing them. Some sources do not consider these to be true color blindness, because the failure is of perception, not of vision. They are forms of visual agnosia. The most common type of color blindness is red-green color blindness. This when one is unable to or has difficulty distinguishing red and green hues because of the absence or mutation of the red or green retinal photoreceptors. This type is part of the dichromacy color blindness. This when someone can match any color they see with some mixture of just two primary colors. This is different from people with fully functioning color perception that require all three primary colors. Because of this, certain pairs of colors, like red and green, that seem quite different to the normal viewer might appear to be the same color to someone with this type of color blindness. Dichromats often have difficulty with daily tasks like picking out clothes to wear or distinguishing between the colors of a stoplight. The final type is anomalous trichromacy. People with this type require different color matches from people with normal vision. For example, in order to match a given spectral yellow light, they may need more red light or green light in a red/green mixture than a normal viewer.
Color blindness has been cured in monkeys using gene therapy, but this will not be available to humans until it can be proven to be safe. The only other treatment available for color blindness is filtered lenses that enhance color perception. These lenses can be difficult to find, though, as they are only available from a limited number of eye care practitioners. It is mostly up to the affected person to adjust their lives accordingly with the condition. One may need to memorize the order of stop lights rather than the colors or organize their clothes in a way that will prevent them from accidentally put on clashing colors.

Terms: Color blindness, Cones, Achromatopsia, Photoreceptors, Monochromacy, Dichromacy, Anomalous Trichromacy, Perception

https://www.youtube.com/watch?v=8Aaivktz8G0 I used this source, because it had all of the basic background information on the topic and presented it in an easy to process manner.
http://en.wikipedia.org/wiki/Color_blindness I used this source, because it had a lot of technical information that went further into depth than many of the other sources I found.
http://www.allaboutvision.com/conditions/colordeficiency.htm I used this source, because it focused more on how people with color blindness are affected by the condition, rather than just the technical information about the condition itself.

(1a) Gender Differences in Color Blindness and the Visual System

(1b) Color blindness was discussed in the chapter but didn’t go into depth on gender differences.

(1c) I was interested in this topic because males and females are quite active now a days providing opinions on why their counterpart, whether male or female are better equipped in society both physically or mentally.

(2) Color blindness, or color vision deficiency, is the inability or decreased ability to see color, or perceive color differences, under normal lighting conditions. The most identifiable cause is that the cones that perceive color in light and transmit information to the optic nerve are undeveloped or damaged. This fault in cone development is sex linked, photopigments are carried on the x chromosome which males have one x chromosome and females have two. Which give males a higher probability to have photopigment problems compared to females. Certain causes linked to color blindness include: acquired through drugs or disease, it could be inherited, genetics, brain damage, retinal damage, and much more. Currently there are no treatment options to cure color deficiencies.

Gender differences in male and female are always a popular but yet sensitive topic in today's society. James Owen, a writer for National Geographic, dove into one of these topics called “Men and Women Really Do See Things Differently.” What he found is that men and women really don’t see eye to eye, literally. Studies have shown that females are much better at discriminating among colors, while males are excellent at tracking fast-moving objects and discerning detail from a distance. These evolutionary differences may be rooted to the hunting and gathering past. Researchers delivered the notion that throughout the visible spectrum, males require longer wavelengths than females in order to experience the same hue. Longer wavelengths are associated with warm colors, like red and orange. The neuron development in the visual cortex is said to also contribute to seeing shades of blue, greens and yellows. Mens visual cortex is boosted by hormones also, 25 percent more to be exact, which gives them greater ability to see quick changing details from a far, better than colors. The studies findings go on to support the hunter-gatherer hypothesis, which states that sexes evolved distinct psychological abilities to fit their prehistoric roles. Males being the hunters, and females being the gatherers.

My last source provides some good information in regards to the perception of color and the visual system. Men and women look at different things because they interpret the world differently. No matter what, women and men have their own ways of seeing the beauty of things, sometimes the same might I add. Men examine pictures and the environment using a rapid eye movement, women more so tend to gaze. It is natural for men to look for a threat since it is evolutionary part of their make up. Women on the other hand tend to gaze and acknowledge the more social aspect of a picture or the environment. We both see beauty, we just tend to look at it differently.

(3) Terms : color blindness, color discrimination, color deficiencies, color in visual cortex

(4) http://en.wikipedia.org/wiki/Color_blindness
This source provided credible information that I used to define color blindness, show the causes of color blindness, and the treatments of color blindness. This source provided more thorough information on color blindness than the textbook.

http://news.nationalgeographic.com/news/2012/09/120907-men-women-see-differently-science-health-vision-sex/
This source provided credible information that I used to discuss gender differences in color perception rooted back to the evolutionary idea of hunters and gatherers.

http://www.sciencedaily.com/releases/2012/11/121130222243.htm
This last source was the best I could find, other sources tended to repeat information from my previous sources. So I used the information from this article to explore how men and women view the visual world differently.

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